Efficient Computational Methods
for Drug Design
using Metabolic Pathways

Team:

Description:

The human body at its genetic core consists of enzymes that catalyze reactions. These reactions generate compounds by consuming a set of other compounds. Chemical molecules (drugs) that inhibit enzymes can reduce or increase the generation of one or more compounds. The goal of drug discovery is to derive chemical molecules that inhibit enzymes to have a therapeutic effect by increasing or reducing the generation of more or more target compounds while minimizing the negative effects (side effects) on other important compounds. Currently this is done mainly by testing a large number of molecules to inhibit enzymes using high throughput screening. This is a very costly (more than US $800 million per drug) and inaccurate (90 to 95% of the drugs entering the clinical development fail) process. New strategies to reduce the attrition rates of drugs by considering toxicity as well as efficacy prior to clinical study is sorely needed. Metabolic networks define the relationships between enzymes and compounds. Although many models for metabolic networks have been developed, they have not been used for predicting the efficacy and the side effects of drugs.

We are developing effective computational models and tools that will exploit the structure of the metabolic networks to better understand the impact of molecules on different enzymes as well understanding which enzymes should be inhibited so that the target compounds can be appropriate affected. The proposed work can be broadly divided into the following two tasks:

  1. Development of biologically accurate computational cost models for metabolic networks.
  2. Development of efficient algorithms for identifying target enzymes that can have a desired effect on target compounds. Given a set of target (unwanted) compounds, we will develop computational tools that will identify a subset of enzymes whose inhibition disrupts the production of the target compounds with minimum or close to minimum side effect (i.e., disruption of non-target compounds) as defined by the underlying cost model.

We will validate the accuracy of the proposed models and the search methods using successful and failed drugs that have been screened in existing or ongoing preclinical trials.

Publications:

  • Padmavati Sridhar, Tamer Kahveci, Sanjay Ranka,An iterative algorithm for metabolic network-based drug target identification, Proceedings of  PSB 2007. (Abstract) (PDF)

Last updated:June 23, 2005 10:28 EDT